Microbiology: Bacterial Structure, Function, and Pathogenicity
Termini in questo insieme (29)
Staphylococci describes bacteria arranged in grape-like clusters.
Leads to loss of structural connection between the outer membrane and peptidoglycan layer.
Capsule/slime layer production directly contributes to biofilm persistence.
Presence of outer membrane and LPS is most informative.
Capsule protects bacteria from phagocytosis.
Endospore formation enhances survival under starvation.
Likely due to increasing repellent concentration in the environment.
Ribosomes are the best target to inhibit bacterial protein synthesis.
Because the cell wall target is absent in L-forms.
Cells cannot tumble and reorient, impairing chemotaxis.
Because they are naturally spherical and do not need MreB for shape maintenance.
Loss of capsule production reduces pathogenicity.
They have a mycolic acid-rich cell wall that reduces permeability.
Mycolic acid layer is connected to peptidoglycan by arabinogalactan.
Conjugation pilus facilitates gene transfer.
Peptidoglycan-derived molecules trigger inflammation.
Mycobacterium tuberculosis has an acid-fast cell wall.
Reduced barrier protection against antibiotics.
Because of a thick peptidoglycan layer.
O antigen is a surface-exposed target with antigenic variation.
Presence of 70S ribosomes indicates a bacterium.
They can spread antibiotic resistance genes.
Shape changes from rod to coccoid.
Sarcinae describes cube-like packets of eight cells.
Gas vesicles regulate buoyancy.
Incorrect; pili-mediated twitching and gliding motility also occur.
Lipid A is responsible for endotoxic effects.
Outer membrane is unique to Gram-negative bacteria.
Magnetosome helps navigation along magnetic fields.