Which of the following best defines selective toxicity for antiviral drugs?

Why is it generally harder to achieve selective toxicity for antiviral drugs than for antibacterial drugs?

A patient begins an antiviral that reduces their daily productive viral replication by 70% each day (i.e., viral output each day is 30% of the previous day). If the baseline plasma viral load is 1,000,000 copies/mL, what is the expected viral load after 3 days of therapy?

Which explanation best accounts for the selective activity of acyclovir against herpes viruses but not most host cells?

Design a rational combination therapy strategy for chronic HIV infection that uses complementary mechanisms to reduce viral load and delay resistance. Which combination is best and why?