Surgical damage to intestinal lacteals prevents chylomicrons from entering the lymphatic system. Which systemic effect is most likely to follow?

Development of hyperaminoacidemia due to enhanced co-transport of peptides with untransported chylomicrons into systemic veins, increasing plasma protein synthesis and masking any lipid-related symptoms.

Immediate rerouting of chylomicrons through the portal vein to the liver with no physiological consequence because the vascular endothelium permits chylomicron-sized particles to pass freely into portal circulation.

Decreased delivery of dietary long-chain triglyceride–derived lipids to peripheral tissues via chylomicrons, resulting in reduced postprandial plasma chylomicron concentrations and impaired peripheral lipid availability; this can cause fat malabsorption symptoms despite intact micelle formation and enterocyte reassembly.

A compensatory increase in biliary bile salt secretion that dissolves chylomicrons in the lumen and allows absorption of intact lipoproteins directly into the systemic arterial circulation via the intestinal brush border.