Assess the impact of autophosphorylation on receptor tyrosine kinases in the context of insulin signaling.

Autophosphorylation leads to the degradation of receptor tyrosine kinases, terminating the signal.

Autophosphorylation has no impact on receptor tyrosine kinases or signaling.

Autophosphorylation inhibits receptor tyrosine kinases, preventing further signaling.

Autophosphorylation activates receptor tyrosine kinases, enabling them to phosphorylate downstream targets and propagate the signal.