Indicate which statements are true. Correct all false statements by changing the underlined words.
__________ In phagocytosis, adhesion involves the binding between complementary chemicals on a phagocyte and on the membrane of a body cell.

Verified step by step guidance

1. Step 1: Understand the key terms in the statement. 'Phagocytosis' is the process by which certain cells (phagocytes) engulf and digest particles such as bacteria or cellular debris.
2. Step 2: Focus on the term 'adhesion' in the context of phagocytosis. Adhesion refers to the initial attachment of the phagocyte to the target particle or microorganism.
3. Step 3: Analyze the phrase 'binding between complementary chemicals on a phagocyte and on the membrane of a body cell.' In phagocytosis, adhesion actually involves binding between receptors on the phagocyte and molecules on the surface of the target particle (such as a bacterium), not a 'body cell.'
4. Step 4: Correct the false part by replacing 'body cell' with 'microorganism' or 'target particle' to accurately describe the interaction during phagocytosis.
5. Step 5: Summarize the corrected statement: In phagocytosis, adhesion involves the binding between complementary chemicals on a phagocyte and on the membrane of a microorganism (or target particle).

Key Concepts

Phagocytosis

Phagocytosis is a cellular process where specialized cells, called phagocytes, engulf and digest foreign particles, microbes, or dead cells. It is a key mechanism in the immune response to remove pathogens and debris from the body.

Adhesion in Phagocytosis

Adhesion is the initial step in phagocytosis where phagocytes recognize and bind to specific molecules on the surface of target particles, such as pathogens, not body cells. This binding is mediated by receptors on the phagocyte that detect complementary ligands on the target.

Target Recognition in Phagocytosis

Phagocytes bind to foreign particles or pathogens, not normal body cells, during phagocytosis. The recognition involves complementary chemical signals like opsonins on the pathogen surface, which facilitate binding and subsequent engulfment.